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Eliezer Monge is accused of inappropriately touching an 18-year-old patient. (Farmington Public Safety) A Detroit doctor has been charged with assault and battery after an 18-year-old patient claimed he touched her inappropriately and attempted to kiss her while alone in the exam room. The alleged assault between Dr. Eliezer Monge and the unidentified patient occurred in July, The Detroit Free Press reported. The actions of [Monge] in this case are reprehensible, Frank Demers, Farmington Public Safety director, told the newspaper. The victim came to him for help, and sadly he took advantage of her in a manner that is criminal and immoral. The patient accused Monge of instructing her to lie on her back on the examination table before he began touching her inappropriately. He allegedly then tried to kiss her on the mouth when she sat up, but she turned away and hips mouth landed on her neck, The Detroit Free Press reported. More on this…
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In a poster check these guys out presentation titled, Inducible MyD88/CD40 (iMC) Costimulation Provides Ligand-Dependent Tumor Eradication By CD123-Specific Chimeric Antigen Receptor T Cells, Bellicum scientists targeted CD123, which is highly expressed in acute myeloid leukemia (AML) and leukemic stem cells. Results demonstrated that GoCAR-T removed CD123-positive leukemic cells in animal models through rimiducid-activated costimulation. Conversely, infrequent costimulation with rimiducid led to reduced activity of CAR T cells, supporting the technologys potential to find more info provide control over the this link activation, expansion and persistence of cells to achieve a desired level of safety and anti-tumor potency. Additional data were presented on the Companys GoTCR technology, which also uses an iMC costimulatory switch. The presence of GoTCR and rimiducid triggers the release of cytokines that upregulate MHC (major histocompatibility complex) on tumor cells, exposing them to potent immune response by both engineered and endogenous T cells. In the study outlined in a poster presentation titled, Inducible MyD88/CD40 (iMC) Enhances Proliferation and Survival of Tumor-Specific TCR-Modified T Cells and Improves Anti-Tumor Efficacy in Myeloma, T cells were engineered to express tumor antigen-specific T-cell receptors (TCRs) targeting preferentially-expressed antigen in melanoma (PRAME) or Bob1. PRAME is overexpressed in a wide variety of cancers including melanoma, sarcoma and several types of leukemias. Bob1 is also found to be highly expressed in certain leukemias, along with lymphomas and myelomas. Results demonstrated that the rimiducid-driven iMC costimulatory switch provided potent T-cell activation, proliferation and persistence, synergizing with signals from PRAME- or Bob1-targeted TCRs for improved anti-tumor efficacy in vitro and in vivo.
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